JA
Jane Antony
  • Post-Doc, Stanford University
Research fields
  • Cancer biology
Personal information

Education

PhD, Imperial College London, 2017

Lab information

Michael Clarke Lab
https://profiles.stanford.edu/jane-antony

Research focus

Cancer Biology, Cell Signalling, Signal Transduction pathways, Stem cells, Cancer Stem Cells, Epithelial-to-Mesenchymal Transition (EMT), Metastasis

Publications

1. The GAS6-AXL signaling network is a mesenchymal (Mes) molecular subtype – specific therapeutic target for ovarian cancer. Antony J, et al. | Sci. Signal. 9, ra97 (2016)
Focus Piece: New twists in the AXL(e) of tumor progression. Halmos B, and Haura EB | Vol. 9, Issue 448, pp. fs14

2. AXL-driven EMT state as a targetable conduit in cancer. Antony J, Huang RYJ | Cancer Res 2017; 77(14): 3725-32

3. Targeting the AXL signaling pathway in ovarian cancer. Huang RJY, Antony J, Tan TZ, Tan DSP | Mol Cell Oncol 2017; 4(2): e1263716

4. The Tumor-Suppressor Protein OPCML Potentiates Anti–EGFR- and Anti–HER2-Targeted Therapy in HER2-Positive Ovarian and Breast Cancer. Zanini E, Louay L, Antony J, et al. | Mol Cancer Ther 2017; 16(10): 2246-56 | MCT-17-0081.

5. The phosphatase PTPRG inactivates the receptor tyrosine kinase AXL sequestered in plasma-membrane lipid domains by the tumour suppressor OPCML. Antony J, et al. | EMBO Reports (2018) e45670
News and Views: Synergistic inactivation of AXL: a (cross)road to cure ovarian cancer? Zurzolo C | EMBO Reports (2018) e46492

6. Modulation of Wnt signaling in primary tissues through Cdkn2a regulation. Adorno M, Robilant B, Acosta VH, Sikandar S, Antony J, et al. 2018 Scientific Reports

7. Sensitization of cancer cells via non‐viral delivery of apoptosis inducing proteins using a cationic bolaamphiphile
K Narayanan, M Khan, B Gopalan, J Antony, T Das, YY Yang, ACA Wan
Biotechnology journal, 1800020
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