Education

M.D., Medical School, Karolinska Institutet, Stockholm, Sweden, 2014

Current position

M.D./Ph.D. student, Center for Infectious Medicine, Department of Medicine Huddinge, Karolinska Institutet, Stockholm, Sweden (2010-present)

Publications

1. Theorell, J., Schlums, H., Chiang, S. C., Huang, T. Y., Tattermusch, A., Wood, S. M. and Bryceson, Y. T. (2011). Sensitive and viable quantification of inside-out signals for LFA-1 activation in human cytotoxic lymphocytes by flow cytometry. J Immunol Methods 366(1-2): 106-118.
   
2. Bryceson, Y. T., Chiang, S. C., Darmanin, S., Fauriat, C., Schlums, H., Theorell, J. and Wood, S. M. (2011). Molecular mechanisms of natural killer cell activation. J Innate Immun 3(3): 216-226.
   
3. Chiang, S. C.*, Theorell, J.*, Entesarian, M., Meeths, M., Mastafa, M., Al-Herz, W., Frisk, P., Gilmour, K. C., Ifversen, M., Langenskiold, C., Machaczka, M., Naqvi, A., Payne, J., Perez-Martinez, A., Sabel, M., Unal, E., Unal, S., Winiarski, J., Nordenskjold, M., Ljunggren, H. G., Henter, J. I. and Bryceson, Y. T. (2013). Comparison of primary human cytotoxic T-cell and natural killer cell responses reveal similar molecular requirements for lytic granule exocytosis but differences in cytokine production. Blood 121(8): 1345-1356. *These authors have contributed equally to this work
   
4. Hagberg, N., Theorell, J., Eloranta, M. L., Pascal, V., Bryceson, Y. T. and Ronnblom, L. (2013). Anti-NKG2A autoantibodies in a patient with systemic lupus erythematosus. Rheumatology (Oxford) 52(10): 1818-1823.
   
5. Hagberg, N., Theorell, J., Schlums, H., Eloranta, M. L., Bryceson, Y. T. and Ronnblom, L. (2013). Systemic lupus erythematosus immune complexes increase the expression of SLAM family members CD319 (CRACC) and CD229 (LY-9) on plasmacytoid dendritic cells and CD319 on CD56(dim) NK cells. J Immunol 191(6): 2989-2998.
   
6. Saghafian-Hedengren, S., Sohlberg, E., Theorell, J., Carvalho-Queiroz, C., Nagy, N., Persson, J. O., Nilsson, C., Bryceson, Y. T. and Sverremark-Ekstrom, E. (2013). Epstein-Barr virus coinfection in children boosts cytomegalovirus-induced differentiation of natural killer cells. J Virol 87(24): 13446-13455.