1. Pal K, Nowak R, Billington N, Li R, Ghosh A, Sellers J, Fowler V. (2020). Megakaryocyte migration defects due to nonmuscle myosin IIA mutations underly thrombocytopenia in MYH9-Related Disease. Blood.
https://doi.org/10.1182/blood.2019003064.
2. Smith AS, Pal K, Nowak RB, Demenko A, Da Costa L, Favier R, Pecci A, Fowler V. (2019). MYH9-related disease mutations cause abnormal red blood cell morphology through increased myosin-actin binding at the membrane. Am J Hematol. 94(6):667-766
3. Nichols H, Yee M, Polley D, Saifeddine M, Lee K, Pal K, Daines M, Wilson E, Boitano S, Hollenberg M, DeFea K. (2017). Protease-activated-Receptor-2-Induced signaling through β-arrestin-2 Mediates Alternaria Serine Protease-induced Asthma. (In review)
4. Mukhopadhyay S, Badgandi H, Hwang SH, Somatilaka B, Shimada I, Pal K. (2017). Trafficking to the primary cilium membrane. Mol Biol Cell. 28(2): 233-239.
5. Pal K, Hwang SH, Somatilaka B, Badgandi H, Jackson P, Defea K, Mukhopadhyay S. (2015). Smoothened and β-arrestins cooperate to determine steady state and sonic hedgehog mediated removal of the G-protein- coupled receptor Gpr161 from the primary cilium. J Cell Biol.
212(7): 861-75 *Article in focus by Ben Short.http://jcb.rupress.org/content/early/2016/03/21/jcb.2127iti3.1
6. Pal K, Badgandi H, Mukhopadhyay S. (2015). Studying G-protein-coupled receptors: Immunoblotting, immunoprecipitation, phosphorylation, surface labeling, and cross-linking protocols. Methods Cell Biol. 127:303-22. doi: 10.1016/bs.mcb.2014.12.003
7. Pal K, Mukhopadhyay S. (2015). Primary Cilium and sonic hedgehog signaling during neural tube patterning: Role of GPCRs and second messengers. Developmental Neurobiology. 75(4): 337-48. doi: 10.1002/dneu.22193.
8. Mittal N, Roberts K, Pal K, Bentolila LA, Fultz E, Minasyan A, Cahill C, Pradhan A, Conner D, Defea K, Evans C, Walwyn W. (2013). Select G-Protein-Coupled Receptors Modulate Agonist-Induced Signaling via a ROCK, LIMK, and β-Arrestin 1 Pathway. Cell Rep. 5(4): 1010-21.
9. Pal K, Mathur M, Kumar P, DeFea K. (2013). Divergent β-arrestin-dependent signaling events are dependent upon sequences within G-protein-coupled-receptor C-termini. J Biol Chem. 288(5): 3265-74.