Ivan Lalanda Salazar
  • CNC-Center for Neuroscience and Cell Biology, University of Coimbra, Portugal, Portugal,
Research fields
  • Neuroscience
Personal information

Education

PhD, Neuroscience and Disease, University of Coimbra, Portugal, 2017

Current position

Post-Doc, Growth Factor Signalling and Brain Ischemia, Center for Neuroscience and Cell Biology, Portugal

Publications

  1. Salazar, I. L., Caldeira, M. V., Curcio, M. and Duarte, C. B. (2016). The Role of Proteases in Hippocampal Synaptic Plasticity: Putting Together Small Pieces of a Complex Puzzle. Neurochem Res 41(1-2): 156-182.
  2. Curcio, M., Salazar, I. L., Mele, M., Canzoniero, L. M. and Duarte, C. B. (2016). Calpains and neuronal damage in the ischemic brain: The swiss knife in synaptic injury. Prog Neurobiol 143: 1-35.
  3. Curcio, M., Salazar, I. L., Inacio, A. R., Duarte, E. P., Canzoniero, L. M. and Duarte, C. B. (2015). Brain ischemia downregulates the neuroprotective GDNF-Ret signaling by a calpain-dependent mechanism in cultured hippocampal neurons. Cell Death Dis 6: e1645.
  4. Leal, G., Afonso, P. M., Salazar, I. L. and Duarte, C. B. (2015). Regulation of hippocampal synaptic plasticity by BDNF. Brain Res 1621: 82-101.
  5. Caldeira, M. V., Salazar, I. L., Curcio, M., Canzoniero, L. M. and Duarte, C. B. (2014). Role of the ubiquitin-proteasome system in brain ischemia: friend or foe? Prog Neurobiol 112: 50-69.
  6. Caldeira, M. V., Curcio, M., Leal, G., Salazar, I. L., Mele, M., Santos, A. R., Melo, C. V., Pereira, P., Canzoniero, L. M. and Duarte, C. B. (2013). Excitotoxic stimulation downregulates the ubiquitin-proteasome system through activation of NMDA receptors in cultured hippocampal neurons. Biochim Biophys Acta 1832(1): 263-274.
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