Lab Manager/Molecular Biologist, Department of Obstetrics and Gynecology, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA
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Malik, M., Britten, J., Cox, J., Patel, A. and Catherino, W. H. (2016). Gonadotropin-releasing hormone analogues inhibit leiomyoma extracellular matrix despite presence of gonadal hormones. Fertil Steril 105(1): 214-224.
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Patel, A., Malik, M., Britten, J., Cox, J. and Catherino, W. H. (2014). Alternative therapies in management of leiomyomas. Fertil Steril 102(3): 649-655.
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Malik, M., Britten, J., Segars, J. and Catherino, W. H. (2014). Leiomyoma cells in 3-dimensional cultures demonstrate an attenuated response to fasudil, a rho-kinase inhibitor, when compared to 2-dimensional cultures. Reprod Sci 21(9): 1126-1138.
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Levy, G., Malik, M., Britten, J., Gilden, M., Segars, J. and Catherino, W. H. (2014). Liarozole inhibits transforming growth factor-beta3--mediated extracellular matrix formation in human three-dimensional leiomyoma cultures. Fertil Steril 102(1): 272-281 e272.
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Britten, J. L., Malik, M., Levy, G., Mendoza, M. and Catherino, W. H. (2012). Gonadotropin-releasing hormone (GnRH) agonist leuprolide acetate and GnRH antagonist cetrorelix acetate directly inhibit leiomyoma extracellular matrix production. Fertil Steril 98(5): 1299-1307.
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Gilden, M., Malik, M., Britten, J., Delgado, T., Levy, G. and Catherino, W. H. (2012). Leiomyoma fibrosis inhibited by liarozole, a retinoic acid metabolic blocking agent. Fertil Steril 98(6): 1557-1562.
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McCarthy-Keith, D. M., Malik, M., Britten, J., Segars, J. and Catherino, W. H. (2011). Gonadotropin-releasing hormone agonist increases expression of osmotic response genes in leiomyoma cells. Fertil Steril 95(7): 2383-2387.
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Malik, M., Norian, J., McCarthy-Keith, D., Britten, J. and Catherino, W. H. (2010). Why leiomyomas are called fibroids: the central role of extracellular matrix in symptomatic women. Semin Reprod Med 28(3): 169-179.
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Malik, M., Webb, J. and Catherino, W. H. (2008). Retinoic acid treatment of human leiomyoma cells transformed the cell phenotype to one strongly resembling myometrial cells. Clin Endocrinol (Oxf) 69(3): 462-470.
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Gonzales, E., Chen, H., Munuve, R., Mehrani, T., Britten-Webb, J., Nadel, A., Alam, H. B., Wherry, D., Burris, D. and Koustova, E. (2006). Valproic acid prevents hemorrhage-associated lethality and affects the acetylation pattern of cardiac histones. Shock 25(4): 395-401.
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Lin, T., Alam, H. B., Chen, H., Britten-Webb, J., Rhee, P., Kirkpatrick, J. and Koustova, E. (2006). Cardiac histones are substrates of histone deacetylase activity in hemorrhagic shock and resuscitation. Surgery 139(3): 365-376.
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Jaskille, A., Koustova, E., Rhee, P., Britten-Webb, J., Chen, H., Valeri, C. R., Kirkpatrick, J. R. and Alam, H. B. (2006). Hepatic apoptosis after hemorrhagic shock in rats can be reduced through modifications of conventional Ringer's solution. J Am Coll Surg 202(1): 25-35.
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Seldin, M. F., Morse, H. C., 3rd, D'Hoostelaere, L., Britten, J. L. and Steinberg, A. D. (1987). Mapping of alpha-spectrin on distal mouse chromosome 1. Cytogenet Cell Genet 45(1): 52-54.
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Steinberg, A. D., Klinman, D. M., Kastner, D. L., Seldin, M. F., Gause, W. C., Scribner, C. L., Britten, J. L., Siegel, J. N. and Mountz, J. D. (1987). Genetic and molecular genetic studies of murine and human lupus. J Rheumatol Suppl 14 Suppl 13: 166-176.