Mutsuhiro Takekawa Division of Cell Signaling and Molecular Medicine, Institute of Medical Science, The University of Tokyo, JAPAN, JAPAN,
1 protocol

Ko Fujioka Division of Cell Signaling and Molecular Medicine, Institute of Medical Science, The University of Tokyo, JAPAN, JAPAN,
1 protocol

Yuji Kubota
  • Division of Cell Signaling and Molecular Medicine, Institute of Medical Science, The University of Tokyo, JAPAN, JAPAN,
  • 1 Author merit

Education

Ph.D., Graduate School of Science, The University of Tokyo, Japan, 2011

Current position

Assistant Professor, Division of Cell Signaling and Molecular Medicine, Institute of Medical Science, The University of Tokyo, Japan

Publications

  1. Iijima M, Kubota Y, Sawa R, Kubota Y, Hatano M, Igarashi M, Kawada M, Momose I, Takekawa M and Shibasaki M. A guanine derivative as a new MEK inhibitor produced by Streptomuces sp. MK63-43F2. J Antibiot. doi.org/10.1038/ja.2017.100 (2017) 
  2. Kubota Y, O’Grady P, Saito H and Takekawa M. Oncogenic Ras abrogates MEK SUMOylation that suppresses the ERK pathway and cell transformation. Nature Cell Biol. 13, 282-291 (2011)
1 Protocol published
Wheat Germ Agglutinin (WGA)-SDS-PAGE: A Novel Method for the Detection of O-GlcNAc-modified Proteins by Lectin Affinity Gel Electrophoresis
Authors:  Ko Fujioka, Yuji Kubota and Mutsuhiro Takekawa, date: 12/05/2018, view: 125, Q&A: 0
Diverse cytoplasmic and nuclear proteins dynamically change their molecular functions by O-linked β-N-acetylglucosamine (O-GlcNAc) modification on serine and/or threonine residues. Evaluation of the O-GlcNAcylation level of a specific protein, ...
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