Harriet Rea Dept Neurology, Baylor College of Medicine, USA
1 protocol

J. Regino Perez-Polo Dept Biochem. & Mol. Biol., University of Texas Medical Branch, USA
1 protocol

Smitha K. Infante
  • Dept Biochem. & Mol. Biol., University of Texas Medical Branch, USA
Research focus
  • Neuroscience
  • 1 Author merit


Ph.D. in Cell Biology, with an emphasis in neurotoxicology, Department of Biochemistry and Molecular Biology, University of Texas Medical Branch, Galveston, Texas, 2012

Current position

Medical Writer II, Medpace, Inc. (06/2015-present)


  1. Infante, S. K., Oberhauser, A. F. and Perez-Polo, J. R. (2013). Bax phosphorylation association with nucleus and oligomerization after neonatal hypoxia-ischemia. J Neurosci Res 91(9): 1152-1164.
  2. Infante, S. K., Rea, H. C. and Perez-Polo, J. R. (2013). Transgenerational effects of neonatal hypoxia-ischemia in progeny. Int J Dev Neurosci 31(6): 398-405.
  3. Perez-Polo, J. R., Rea, H. C., Johnson, K. M., Parsley, M. A., Unabia, G. C., Xu, G., Infante, S. K., Dewitt, D. S. and Hulsebosch, C. E. (2013). Inflammatory consequences in a rodent model of mild traumatic brain injury. J Neurotrauma 30(9): 727-740.
  4. Bhat, K. M., Gaziova, I. and Krishnan, S. (2007). Regulation of axon guidance by slit and netrin signaling in the Drosophila ventral nerve cord. Genetics 176(4): 2235-2246.
1 Protocol published
Authors:  J. Regino Perez-Polo, Harriet C. Rea and Smitha K. Infante, date: 11/20/2015, view: 7301, Q&A: 0
Neonatal hypoxia-ischemia (HI) affects 60% of low birth weight infants and up to 40% of preterm births. Cell death and brain injury after HI have been shown to cause long-lasting neurological deficits. Two motor coordination tests on rats that had ...
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