All patients underwent a preoperative overnight sleep study at home or in the hospital using a type 3 HSAT (ApneaLink Plus; ResMed, San Diego, CA). It includes a nasal pressure transducer which measure flow limitation and snoring, and records on a 16-bit signal processor and a sampling rate of 100 Hz. In addition to HSAT, oxyhemoglobin saturation was simultaneously monitored using high-resolution pulse oximetry wristwatch (PULSOX-300i, Konica Minolta Sensing, Inc, Osaka, Japan). The oxygen probe of the oximetry was attached to finger of the nondominant hand. The sampling frequency was set as 1 Hz with an averaging time of 3s. The resolution was 0.1%. All patients breathed room air during recording.
The data was extracted the next morning using ApneaLink and Profox (Profox Associates, Escondido, California, USA) software, respectively. All data was processed by a technician blinded to the clinical data and the STOP-Bang score. The sleep parameters variables were extracted from the ApneaLink Plus including the AHI. The sleep-associated apnea and hypopnea events were scored according to the American Academy of Sleep Medicine criteria. Apnea was defined as airflow reduction of ≥90% for ≥10s from baseline. Hypopnea was defined as reduction in airflow for ≥30% for ≥10s from baseline and associated with ≥3% oxyhemoglobin desaturation [14]. Patients with an AHI ≥15 events per hour were considered to have moderate-to-severe OSA, and those with AHI ≥30 events per hour were considered to have severe OSA.
ODI, duration oxygen saturation (SpO2) <90%, lowest, and average SpO2 were extracted from oximetry using Profox software. ODI is defined as the number of events per hour with at least 4% decrease in saturation from the average saturation in the preceding 120s for at least 10s [10]. The oximetry recording data were processed which was recorded between 00:00 to 6:00 hours of night, although it was not known if patients were asleep during this entire period.
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