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We tested the hypothesis that genomic disruption of the Avpr1a gene results in a lack of specific Avpr1a binding in the brain in M and F hamsters (Avpr1a KO: 3 M, 3 F; heterozygote: 3 M, 3 F; and WT: 3 M, 2 F). Binding densities for Avpr1a and Oxtr were analyzed by using 2 (sex) × 3 (genotype) ANOVAs. Appropriate Fisher’s least significant difference (LSD) post hoc tests were done to probe significant main effects and interactions.
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